Search results for " Hypertrophic"

showing 10 items of 19 documents

Ultrasonography (US) in the assessment of pediatric non traumatic gastrointestinal emergencies

2013

AbstractBackgroundNon traumatic gastrointestinal emergencies in the children and neonatal patient is a dilemma for the radiologist in the emergencies room and they presenting characteristics ultrasound features on the longitudinal and axial axis. The most frequent emergencies are : appendicitis, intussusceptions, hypertrophic pyloric stenosis, volvulus due to intestinal malrotation. The aim of this article is to familiarize the reader with the US features.MethodsA retrospective analysis of 200 ultrasound examinations performed in neonatal and children patients with fever, abdominal pain, leukocytosis, vomiting and diarrhea were evaluated.ResultsOf 200 exame 50 cases of intussusceptions, 100…

Abdominal painmedicine.medical_specialtygastrointestinal pediatric emergenciesRadiological and Ultrasound Technologymedicine.diagnostic_testbusiness.industryResearchInterventional radiologyVolvulusmedicine.diseaseAppendicitisHypertrophic pyloric stenosisAppendicitisVolvulusIntestinal malrotationmedicineVomitingGastrointestinal pediatric emergencies intussusceptionsintussusceptionsAppendicitis; Gastrointestinal pediatric emergencies intussusceptions; Hypertrophic pyloric stenosis; VolvulusRadiologymedicine.symptomAbscessbusinessHypertrophic Pyloric Stenosis
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Release kinetics of early ischaemic biomarkers in a clinical model of acute myocardial infarction

2014

ObjectiveTo determine the release kinetics of different biomarkers with potential as novel early ischaemic biomarkers in patients with acute coronary syndrome (ACS); it is difficult to establish the detailed release kinetics in patients with acute myocardial infarction (AMI).MethodsWe analysed the release kinetics of soluble fms-like tyrosine kinase (sFlt-1), ischaemia modified albumin (IMA), and heart-type fatty acid binding protein (hFABP) in patients with hypertrophic obstructive cardiomyopathy who were undergoing transcoronary ablation of septal hypertrophy (TASH), a procedure mimicking AMI. Consecutive patients (n=21) undergoing TASH were included. Blood samples were collected before T…

Ablation TechniquesMalemedicine.medical_specialtyAcute coronary syndromeMyocardial InfarctionCardiomyopathySerum albuminSerum Albumin HumanFatty Acid-Binding ProteinsFatty acid-binding proteinPredictive Value of TestsInternal medicineHumansMedicineMyocardial infarctionSerum AlbuminAgedImmunoassayVascular Endothelial Growth Factor Receptor-1biologybusiness.industryCardiomyopathy HypertrophicMiddle Agedmedicine.diseaseUp-RegulationKineticsEarly DiagnosisPredictive value of testsbiology.proteinCardiologyFemaleFatty Acid Binding Protein 3Myocardial infarction diagnosisCardiology and Cardiovascular MedicinebusinessFatty Acid Binding Protein 3BiomarkersHeart
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GAA trinucleotide repeat expansion in variant Friedreich's ataxia families.

1997

Phenotypic variants in Friedreich's ataxia include late onset, preservation of the lower limbs tendon reflexes, and slow progression. We describe clinical and electrophysiological features from three families with Friedreichlike phenotypes. Friedreich's ataxia diagnosis was confirmed by finding two allelic expansions of the GAA trinucleotide repeat at the X25 gene. In family 1 both patients had a late-onset phenotype with preservation of knee and ankle jerks, lack of cardiomyopathy, and preserved H reflex. One of them did not have electrophysiologic evidence of sensory axonal neuropathy. Patients from family 2 showed variability in the age of onset, and 2 out of 3 affected children had hype…

AdultMaleReflex Stretchcongenital hereditary and neonatal diseases and abnormalitiesPathologymedicine.medical_specialtySensory axonal neuropathyAtaxiaPhysiologyGenetic LinkageAction PotentialsLate onsetBiologyH-ReflexCellular and Molecular NeuroscienceDegenerative diseaseTrinucleotide RepeatsPhysiology (medical)medicineHumansNeurons AfferentChildAllelesLegGenetic VariationDNACardiomyopathy Hypertrophicmedicine.diseasePedigreePeripheral neuropathyFriedreich AtaxiaReflexDisease ProgressionFemaleNeurology (clinical)medicine.symptomAge of onsetTrinucleotide repeat expansionMusclenerve
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Association between polymorphisms of endothelial nitric oxide synthase gene (NOS3) and left posterior wall thickness (LPWT) of the heart in Fabry dis…

2008

Fabry disease is an X-chromosomal storage disorder due to loss-of-function mutations of the GLA gene encoding the lysosomal enzyme α-galactosidase A. Accumulating glycosphingolipid deposits disturb the function of various cells, in particular that of myocytes, arterial smooth-muscle cells, and vascular endothelium. Hypertrophic cardiomyopathy, for example measured by left posterior wall thickness (LPWT) of the heart, represents a major component of Fabry disease morbidity in adult patients. Endothelium-derived nitric oxide (eNO), produced by eNO synthase (eNOS), is a key regulator of vessel wall function and cardiovascular homeostasis. We analysed the effect of the polymorphisms c.894G > T …

AdultMalemedicine.medical_specialtyAdolescentNitric Oxide Synthase Type IIIMutation MissenseMinisatellite RepeatsYoung AdultSex FactorsGene FrequencyInternal medicineGermanyGenotypeGeneticsMedicineHumansGenetic Predisposition to DiseaseAlleleChildAllele frequencyGenetics (clinical)AgedUltrasonographyGeneticsAlpha-galactosidasePolymorphism Geneticbiologybusiness.industryMyocardiumHaplotypeHypertrophic cardiomyopathyNitric Oxide Synthase Type IIIExonsCardiomyopathy HypertrophicMiddle Agedmedicine.diseaseFabry diseaseIntronsEndocrinologyPhenotypeHaplotypesCase-Control Studiesalpha-Galactosidasebiology.proteinFabry DiseaseRegression AnalysisFemaleHypertrophy Left VentricularbusinessJournal of inherited metabolic disease
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Assessment of atrial diastolic function in patients with hypertrophic cardiomyopathy by cine magnetic resonance imaging

2015

Purpose: This study was conducted to assess the role of atrial function by cardiac magnetic resonance (CMR) for the evaluation of diastolic physiology in patients with hypertrophic cardiomyopathy (HCM) compared to healthy controls. Materials and methods: We enrolled 23 consecutive patients affected by HCM and 43 healthy subjects as age-matched control cases (CC). CMR was performed through acquisition of cine steady-state free precession sequences using a 1.5-T scanner. Image postprocessing was carried out using Tracking Tool software. Results: Atrial volumes were significantly higher in patients with HCM compared to CC: maximum atrial volume (p = 0.007) and minimum atrial volume (p = 0.01).…

AdultMalemedicine.medical_specialtyRadiology Nuclear Medicine and ImagingDiastolic functionCardiac magnetic resonanceDiastoleContrast MediaMagnetic Resonance Imaging CineDiastoleInternal medicineImage Interpretation Computer-AssistedmedicineOrganometallic CompoundsHumansIn patientDiastolic functioncardiovascular diseasesHeart AtriaNeuroradiologyOrganometallic Compoundmedicine.diagnostic_testbusiness.industryMedicine (all)UltrasoundAtrial functionHypertrophic cardiomyopathyMagnetic resonance imagingInterventional radiologyGeneral MedicineCardiomyopathy Hypertrophicmedicine.diseaseHypertrophic cardiomyopathyCase-Control Studiescardiovascular systemCardiologyFemalebusinessCase-Control StudieSettore MED/36 - Diagnostica Per Immagini E RadioterapiaHuman
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Ménétrier's disease in a patient with refractory ulcerative colitis: a clinical challenge and review of the literature

2023

Ménétrier’s disease (MD) is a rare disease of the stomach, characterised by hypertrophic gastric folds leading to protein loss. The association with ulcerative colitis (UC) is rare but has been reported in the literature. We report a case of a 29-year-old male affected by UC with an additional diagnosis of MD 3 years after UC diagnosis. UC was refractory to several treatment lines (thiopurines, infliximab, vedolizumab and ustekinumab), and the patient underwent colectomy. Octreotide was administered for MD normalising blood biochemistry, but it was not effective in inducing endoscopic remission of the stomach. Treatment options in patients with MD and UC are discussed.

AdultMalemedicine.medical_specialtymedicine.medical_treatmentGastroenterologyInflammatory bowel diseaseVedolizumabInternal medicineUstekinumabmedicineHumansGastritis HypertrophicColectomybusiness.industryGeneral Medicinemedicine.diseaseUlcerative colitisInfliximabInfliximabMénétrier's diseaseColitis UlcerativeUstekinumabbusinessRare diseasemedicine.drugdrugs: gastrointestinal system; gastroenterology; inflammatory bowel disease
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Sudden Cardiac and Noncardiac Death in Sports: Epidemiology, Causes, Pathogenesis, and Prevention

2018

AbstractAlthough few doubts remain that physical exercise should be widely promoted for maintenance of health and fitness, the risk of adverse events such as sudden death (especially due to cardiac causes, i.e., sudden cardiac death [SCD]) during exercise remains tangible. The overall risk of sudden death in athletes is relatively low (i.e., usually comprised between 0.1 and 38/100,000 person-years), and globally comparable to that of the general population. However, up to 20% of all sudden death cases are still recorded while exercising. The most frequent underlying disorders encountered in SCD are hypertrophic cardiomyopathy and coronary artery disease (CAD), representing three quarters o…

Adultmedicine.medical_specialtyPopulationCardiomyopathysudden deathphysical activityPhysical exerciseCoronary Artery Diseasecardiac arrest030204 cardiovascular system & hematologyRisk AssessmentSudden deathSudden cardiac deathCoronary artery disease03 medical and health sciences0302 clinical medicineRisk Factorsphysical exerciseInternal medicineEpidemiologymedicineHumans030212 general & internal medicineeducationExerciseeducation.field_of_studybusiness.industrysudden death; cardiac arrest; sports; physical exercise; physical activityHypertrophic cardiomyopathyHematologyCardiomyopathy Hypertrophicmedicine.diseaseDeath Sudden CardiacAthletesCardiologysportsCardiology and Cardiovascular MedicinebusinessSeminars in Thrombosis and Hemostasis
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Comparison of Nonclassic and Classic Phenotype of Hypertrophic Cardiomyopathy Focused on Prognostic Cardiac Magnetic Resonance Parameters: A Single-C…

2022

Patients with nonclassic phenotypes (NCP)—more advanced stages of hypertrophic cardiomyopathy (HCM)—constitute an intriguing and heterogeneous group that is difficult to diagnose, risk-stratify, and treat, and often neglected in research projects. We aimed to compare cardiac magnetic resonance (CMR) parameters in NCP versus classic phenotypes (CP) of HCM with special emphasis given to the parameters of established and potential prognostic importance, including numerous variables not used in everyday clinical practice. The CMR studies of 88 patients performed from 2011 to 2019 were postprocessed according to the study protocol to obtain standard and non-standard parameters. In NCP, the late …

Clinical Biochemistrycardiovascular systemcardiovascular diseasesmagnetic resonance imaging; late gadolinium enhancement; hypertrophic cardiomyopathy; phenotype; sudden cardiac death; new imaging techniques; prognosis; outcomes; functional imaging; left ventricular obstruction; left atrium; mitral valve apparatusDiagnostics
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Isolated non-compaction of the myocardium as a cause of coronary and cerebral embolic events in the same patient.

2009

A 44-year-old woman with a history of smoking and previous cerebral thrombo-embolism presented to the emergency department with prolonged chest pain and ECG changes showing an acute anterior myocardial infarction. She was referred to the cath-lab for primary angioplasty. Coronary angiography showed a thrombotic occlusion at the origin of first diagonal …

Coronary angiographyAdultmedicine.medical_specialtyPrimary angioplastyChest painNecrosisVentricular Dysfunction LeftThrombotic occlusionInternal medicineMedicineHumansbusiness.industryCoronary ThrombosisMyocardiumEmergency departmentAcute anterior myocardial infarctionCardiomyopathy HypertrophicCoronary heart diseaseCoronary OcclusionIntracranial EmbolismCirculatory systemCardiologyFemalemedicine.symptomCardiology and Cardiovascular MedicinebusinessCardiomyopathiesMagnetic Resonance AngiographyEuropean heart journal
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Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice

2002

The serine-threonine kinase Akt seems to be central in mediating stimuli from different classes of receptors. In fact, both IGF-1 and IL6-like cytokines induce hypertrophic and antiapoptotic signals in cardiomyocytes through PI3K-dependent Akt activation. More recently, it was shown that Akt is involved also in the hypertrophic and antiapoptotic effects of β-adrenergic stimulation. Thus, to determine the effects of Akt on cardiac function in vivo, we generated a model of cardiac-specific Akt overexpression in mice. Transgenic mice were generated by using the E40K, constitutively active mutant of Akt linked to the rat α-myosin heavy chain promoter. The effects of cardiac-selective Akt overex…

Gene ExpressionTransgenicGlycogen Synthase Kinase 3MiceGSK-3Receptorsgenetics/physiologycytology/metabolismMultidisciplinaryBiological SciencesProtein-Serine-Threonine KinasesDNA-Binding Proteinsenzymology/genetics/pathologyAdrenergicPhosphorylationSignal transductionMitogen-Activated Protein KinasesSignal Transductionmedicine.medical_specialtyCardiomyopathyAnimals; Calcium-Calmodulin-Dependent Protein Kinases; metabolism; Cardiomyopathy; Hypertrophic; enzymology/genetics/pathology; Cell Size; physiology; DNA-Binding Proteins; GATA4 Transcription Factor; Gene Expression; Glycogen Synthase Kinase 3; Mice; Transgenic; Mitogen-Activated Protein Kinases; Myocardial Contraction; Myocardium; cytology/metabolism; Point Mutation; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; Proto-Oncogene Proteins; genetics/physiology; Rats; Receptors; Adrenergic; beta; Signal Transduction; Transcription FactorsMice TransgenicBiologyProtein Serine-Threonine KinasesContractilityIn vivoInternal medicineProto-Oncogene ProteinsReceptors Adrenergic betamedicineAnimalsPoint MutationGlycogen synthaseProtein kinase BPI3K/AKT/mTOR pathwayCell SizeMyocardiumCardiomyopathy HypertrophicMyocardial ContractionGATA4 Transcription FactorRatsEndocrinologyHypertrophicphysiologyCalcium-Calmodulin-Dependent Protein Kinasesbiology.proteinbetametabolismProto-Oncogene Proteins c-aktTranscription Factors
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